Last edited by Malaran
Sunday, April 26, 2020 | History

1 edition of Airway Smooth Muscle in Health and Disease found in the catalog.

Airway Smooth Muscle in Health and Disease

  • 153 Want to read
  • 6 Currently reading

Published by Springer US in Boston, MA .
Written in English

    Subjects:
  • Biochemistry,
  • Life sciences,
  • Animal Physiology

  • Edition Notes

    Statementedited by Ronald F. Coburn
    Classifications
    LC ClassificationsQH345
    The Physical Object
    Format[electronic resource] /
    Pagination1 online resource (334 pages)
    Number of Pages334
    ID Numbers
    Open LibraryOL27015674M
    ISBN 101461307791
    ISBN 109781461307792
    OCLC/WorldCa840281128

    Airway Smooth Muscle in Asthma and COPD Biology and Pharmacology edited by K. Fan Chung. Airway Smooth Muscle in Asthma and COPD provides the clinician with the most up to date information on one of the core physiological processes in airway disease, and offers insights into current and future approaches to management. -heat airways and reduce amount of smooth muscle in the airway -cath goes into airway, press on pedal, heats it and keep doing it until finish whole area of lung -do in severe persistent asthma that is still uncontrolled --> get diff parts of lung and helps symptoms *improves asthma quality of life scores, reduces exacerbations, reduces ER visits.   Relationship Between Smooth Muscle Shortening and Resistance in Isolated Airways, H.W. Mitchell, P.K. McFawn, J.C. Marriott, and M.P. Sparrow Assessment of Airway Smooth Muscle Growth and Division: In Vitro Studies, S.J. Hirst Regulation of Growth of Airway Smooth Muscle by Second Messenger Systems, R.A. Panettieri, :


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Airway Smooth Muscle in Health and Disease by Ronald F. Coburn Download PDF EPUB FB2

I organized this book because there is a need to put together in book form recent advances in our knowledge of how airway smooth muscle:works in Airway Smooth Muscle in Health and Disease book and in disease. After a period when it seemed that progress was very slow, there has been in the past few years an incredibly rapid gathering of knowledge in this : Springer US.

Airway Smooth Muscle in Asthma and COPD is the first book of its kind that focuses entirely on the airway smooth muscle as the prime abnormality in airways diseases such as asthma.

Airways diseases are significant cause of morbidity and mortality across the world, and increasing research into these diseases has developed the understanding of pathophysiological processes in airways disease Format: Hardcover. VIII. References.- 9 Ion Channels in Airway Smooth Muscle.- I.

Introduction.- II. Ion Channels.- III. Patch-Clamp Techniques.- IV. Potassium Channels in Airway Smooth Muscle.- V. Calcium Channels in Airway Smooth Muscle.- VI. Functional Characteristics of Ion Channels in Airway Smooth Muscle.- VII. References.- 10 Coupling Mechanisms in Airway.

I organized this book because there is a need to put together in book form recent advances in our knowledge of how airway smooth muscle:works in health and in disease. After a period when it seemed that progress was very slow, there has been in the past few years an incredibly rapid gathering of knowledge in this area.

The book contains state-of-the-art concepts of plasma membrane receptors, smooth muscle contraction, activation of contractile proteins, and neural signals to the airways.

Of particular value are the chapters that deal with asthmatic hyperreactivity in allergic and exercise-induced : Anthony S. Rebuck. T1 - Regulation of Airway Smooth Muscle Contraction in Health and Disease. AU - Lam, Maggie. AU - Lamanna, Emma. AU - Bourke, Jane E. PY - /6/ Y1 - /6/ N2 - Airway smooth muscle (ASM) extends from the trachea throughout the bronchial tree to the terminal by: 1.

AIRWAY SMoafH MUSCLE IN HEALTH AND DISEASE. Edited by RONALD F. COBURN. New York: Plenum Press,pp, $ Seventeenworld-classinvestigatorsin thefield ofsmooth musclephysiology have contributed chapters to this excellent text.

There has been a rapid increase ofknowledge concerning airway smooth muscle in recent years. Get this from a library. Airway Smooth Muscle in Health and Disease.

[Ronald F Coburn] -- I organized this book because there is a need to put together in book form recent advances in our knowledge of how airway smooth muscle:works in health and in disease. After a period when it seemed. Airway Smooth Muscle in Health and Disease book   Abstract.

Airway smooth muscle (ASM) extends from the trachea throughout the bronchial tree to the terminal bronchioles. In utero, spontaneous phasic contraction of fetal ASM is critical for normal lung development by Airway Smooth Muscle in Health and Disease book intraluminal fluid movement, ASM differentiation, and release of key growth contrast, phasic contraction appears to be absent in the adult lung, and Cited by: 1.

Airways Smooth Muscle: Structure, Innervation and Neurotransmission (Respiratory Pharmacology and Pharmacotherapy): Medicine & Health Science Books @ Request PDF | Regulation of Airway Smooth Muscle Contraction in Health and Disease | Airway Smooth Muscle in Health and Disease book smooth muscle (ASM) extends from the trachea throughout the bronchial tree to the terminal bronchioles.

In asthma and chronic obstructive pulmonary disease (COPD), bronchoconstriction evoked by smooth muscle shortening promotes airway obstruction, a hallmark of asthma and COPD.

Because increases Airway Smooth Muscle in Health and Disease book cytosolic calcium directly regulate the initiation and the development of ASM contraction, changes in calcium homeostasis may promote bronchial. Dear Colleagues, Airway smooth muscle (ASM) controls an airway diameter and regulates the amount of air that reaches the lungs.

ASM dynamic function is portrayed in the plasticity of ASM cells that switch between contractile and proliferative phenotypes in response to environmental, physiological, and pathological signals. airway smooth muscle cell signaling and function in health and disease Home > ATS Conferences > ATS American Journal of Respiratory and Critical Care MedicineVolume   In this book, leading researchers in medicine and molecular pharmacology explain the cellular mechanisms that control airway smooth muscle.

The means by which these are disrupted in disease, and the pharmacologic strategies by which they may be modified are discussed and future therapeutic interventions are identified. Aimed at specialists in pulmonology, this volume provides the Author: Kian Fan Chung.

Airway remodeling in human chronic airway disease is a concept not yet clearly defined [2]. The principal morphologic features are subepithelial fibrosis, myofibroblast hyperplasia, airway smooth muscle hypertrophy, mucus gland and goblet cell hyperplasia, and epithelial disruption [98].

The mechanisms leading to persistent airway changes are. The Epithelium and Airway Remodelling (Stephen T.

Holgate). The epithelium and airway remodelling. Epithelial injury and impaired repair. Changes to the epithelial laminar reticularis (basal laminar).

Submucosal smooth muscle adventitial deposition of matrix. Airway smooth muscle. Vascular remodelling. Dietary Patterns. Various dietary patterns have been linked to the risk of respiratory disease [].The Mediterranean diet has been found to have protective effects for allergic respiratory diseases in epidemiological studies [].This dietary pattern consists of a high intake of minimally processed plant foods, namely; fruit, vegetables, breads, cereals, beans, nuts and seeds, low to Cited by: Airway Calibre in Health and Disease: The Elsevier, - Medical - pages.

0 Reviews. From inside the book. What people are abnormality aerosol airway calibre airway closure airway hyperreactivity airway narrowing airway obstruction airway resistance airway smooth muscle airways disease allergic alveolar alveolar pressure alveoli.

The ubiquitous nature of the role of calcium as a signaling actor in smooth muscle cells (SMC) associated with the fact that the SMC are major constituents of the airways, are two basic findings that clearly justify the importance of this book edited by Yong-Xiao : Stephane Tanguy.

Chronic obstructive pulmonary disease (COPD) is a multi-component disease characterised by airflow limitation and airway inflammation. Exacerbations of COPD have a considerable impact on the quality of life, daily activities and general well-being of patients and are a great burden on the health system.

Alterations in smooth muscle cell function and phenotype contribute to tissue remodeling in various pathologies including obstructive lung (e.g., asthma) and vascular (e.g., atherosclerosis) diseases. The extracellular matrix (ECM) is a major influence on the biology of smooth muscle cells, being an important support structure that provides signaling cues through its biochemical and Author: Michael Schuliga.

At 6–8 wk of gestation, airway smooth muscle cells appear in the trachea and main and lobar bronchi. Muscle then develops sequentially along segmental, terminal, and respiratory bronchioles and alveolar ducts.

The human fetal airway smooth muscle contracts spontaneously in the first trimester; it also responds to pharmacologic manipulation Cited by: F1: Schematic diagram depicting the role of airway smooth muscle (ASM) cell-derived mediators in airway inflammation.

The release of cytokines, chemokines or growth factors (open arrows) can result in ASM as well as inflammatory cell proliferation (dark grey arrows), or the recruitment (light grey arrows) or activation (black arrows) of various cells in the airways. Calcium, a multitasking signaling actor in airway smooth muscle cells, as a target of novel strategies to limit airway disease.

Stephane Tanguy*University Joseph Fourier-Grenoble 1, CNRS, TIMC-IMAG Laboratory CNRS UMR ,PRETA team, Grenoble, F, France *correspondence: @ book review on:Calcium Signaling In Airway Smooth Muscle Author: Stephane Tanguy.

Pulmonary Biology in Health and Disease was conceived as a companion to a handful of expensive, multivolume textbooks. This is part of the promising trend to publish shorter textbooks on the subjects of lung biology and remodeling.

Whoever is familiar with human biology and the far-reaching consequences of the genome and postgenome revolutions is apt to concede that the centerpiece in.

This book explores the role calcium signaling plays in cellular responses in almost all types of cells including airway smooth muscle cells. This universal signaling may result from extracellular calcium influx and/or intracellular calcium release, which are precisely controlled and regulated by ion channels, exchangers and/or transporters on the plasmalemmal or sarcoplasmic reticulum membrane.

Part III: Human airway smooth muscle function in health and disease Chapter Increased in vitro histamine responses in human small airway smooth muscle from patients with chronic obstructive pulmonary disease. Chapter Relaxation responses of airway smooth muscle from subjects with and without chronic bronchitis and airflow Cited by: 3.

Human airway smooth muscle cells (HASMCs) produce corticosteroid-insensitive IL-8 in response to TNFα or LPS in stable disease stages or bacteria-induced exacerbations, respectively. pmitogen-activated-protein-kinases (p38MAPKs) are alternative therapeutic : Jürgen Knobloch, David Jungck, Juliane Kronsbein, Erich Stoelben, Kazuhiro Ito, Andrea Koch.

With increasing disease duration irreversible airway restriction develops from inflammation- driven fibro-proliferative airway remodeling that includes increased deposition of extracellular matrix, the accumulation of airway smooth muscle, and increased numbers of by: Airway smooth muscle hypertrophy and hyperplasia lead to an increase in airway wall thickness.

The pathologic changes above translates to the following physiologic consequences: Decreased airway distensibility (stiffer airways) Diminished elastic recoil; Progressive decline in FEV1, and FVC. The mechanisms underlying airway hyperresponsiveness are still unknown but increased contractility of airway smooth muscle may play a role.

This study sought to demonstrate a relationship between. Airway edema. As the disease becomes more persistent and inflammation more progressive, other factors further limit airflow (figure 2–2). These include edema, inflammation, mucus hypersecretion and the formation of inspissated mucus plugs, as well as structural changes including hypertrophy and hyperplasia of the airway smooth muscle.

T1 - Role of parasympathetic nerves and muscarinic receptors in allergy and asthma. AU - Scott, Gregory D. AU - Fryer, Allison. PY - Y1 - N2 - Parasympathetic nerves control the symptoms and inflammation of allergic diseases primarily by signaling through peripheral muscarinic by: It is characterized by spasmodic contraction of airway smooth muscle, WHEEZING, and dyspnea (DYSPNEA, PAROXYSMAL).

Definition (MEDLINEPLUS) Asthma is a chronic disease that affects your airways. Your airways are tubes that carry air in and out of your lungs. If you have asthma, the inside walls of your airways become sore and swollen.

The airway narrowing is REVERSIBLE by using an inhaled medication called salbutamol (also known as “Ventolin” or “Asmol”) – that binds to a receptor on the smooth muscle and causes it to relax – opening up the airways. BRONCHIOLITIS. Bronchiolitis is also a respiratory illness in children – caused by a chest infection.

The "airways" are the bronchial tubes that conduct air from the mouth to the alveoli of the lungs where oxygen is taken up into the blood stream and carbon dioxide is removed. Thus the term "airway disease" or "airway condition" refers to a disorder that narrows the airways and interferes with the smooth passage of air in and out of the lungs.

1. Provides a low-resistance pathway for air flow; resistance is physiologically regulated by changes in contraction of airway smooth muscle and by physical forces acting upon the airways. Defends against microbes, toxic chemicals, and other foreign matter; cilia, mucus, and phagocytes perform this function.

Warms and moistens the air. In the reverse direction, mast cells secrete CCL19 that, through its receptor CCR7, stimulates airway smooth muscle migration and probably contributes to the smooth muscle hyperplasia (Kaur et al.

Thus, airway smooth muscle is partly dependent on mast cells for its survival and enhanced contractility, whereas mast cells are dependent on Cited by: Dr Jane Bourke is a Senior Lecturer in Pharmacology at Monash University, where she leads the Respiratory Pharmacology Group.

She has a long-standing interest in the regulation of smooth muscle function in the lung and cardiovascular system.

LAM and Other Diseases Characterized by Smooth Muscle Pdf and Pdf G. Nabel Smooth Muscle Cells and Restenosis, George Sopko Airway Smooth Muscle in Asthma, Stewart J. Levine and James H. Shelhamer The Role of Smooth Muscle Cells in Primary Pulmonary Hypertension, Mitchell D. Botney Effects of Nitric Oxide and Cyclic GMP on Author: Taylor And Francis.Estrogen effects on human airway smooth muscle involve cAMP and protein kinase A.

American journal of physiology Lung cellular and molecular physiology. ; (10): L PMCID: Sathish V, Abcejo AJ, Thompson MA, Sieck GC, Prakash YS, Pabelick CM.

Caveolin-1 regulation of store-operated Ca2+ influx in human airway smooth muscle.At 10 days after Cl2 exposure ebook airway smooth muscle mass was increased relative to controls, suggestive of airway smooth muscle hyperplasia and there was evidence of airway fibrosis. No increase in goblet cells occurred at any time by: